Gingival Enlargement and it's types
WHAT IS GINGIVAL ENLARGEMENT?
ClASSIFICATION
A] Based on etiologic factors and pathologic changes
I. Inflammatory Enlargement
- Acute
- Chronic
II. Drug-induced enlargement
III. Enlargements associated with systemic diseases or conditions
i) Conditioned enlargement
- Pregnancy
- Puberty
- Plasma cell gingivitis
- Vitamin C deficiency
ii) Systemic disease causing enlargement
- Leukemia
- Granulomatous diseases (Wegener'sg ranulornatosis, sarcoidosis)
IV.) Neoplastic Enlargement
- Benign
- Malignant
V.) False enlargement
For drug induced enlargements, those drugs must replaced by their alternatives by the dentist and can be bought online from home health care products.
B] Based on location and distribution
I.Localized
II Generalized
III. Marginal
IV. Papillary
V. Diffuse
VI. Discrete
GRADING OF GINGIVAL ENLARGEMENT
Bokenkamp & Bohnhorst 1994
Grade 0 -4 :No signs of gingival enlargement
Grade 1-4 :Enlargement confined to IDP
Grade 2 -4 :Enlargement involves IDP &marginal gingiva
Grade 3-4 :Enlargement covers three quarters / more of crown or less.
Gingival overgrowth index- Mc Gaw et at 1987
Grade 0-4 :No overgrowth, feather edge gingival margin
Grade 1-4 :Blunting of gingival margin
Grade 2-4 :Moderate gingival overgrowth (one third crown length)
Grade 3-4 :Marked gingival overgrowth (more than one thirds of crown)
Clinical index for drug induced gingival overgrowth
1. Severe overgrowth profound thickening of gingiva
2. Large portion of the clinical crown is covered
3. Papillae - retractable
4. Probing depth > 3mm
5.buccolingual dimension 3mm
INFLAMMATORY ENLARGEMENT
a. Chronic inflammatory enlargement
Etiology
- Poor oral hygiene
- Malocclusion
- Plaque accumulation & retention
- Anatomical abnormalities
- Orthodontic appliances
- restorations
Clinical Features
- Slight ballooning of IDP & marginal gingiva
- Life preserver shaped bulge
- Smooth , edematous , bleed easily
- Progress- slowly and painlessly
- Pseudopockets
- Discrete sessile or pedunculated tumor like mass
- Interproximal / marginal or attached gingiva
- Slow growing and painless
B. ACUTE INFLAMMATORY ENLARGEMENT
1. GINGIVAL ABSCESS
Etiology
Foreign substances
Clinical features
- Marginal gingiva or IDP
- Red swelling
- Smooth shiny surface
- Fluctuant and pointed with
- a surface orifice
- Expresses purulent exudate
2. PERIODONTAL ABSCESS (Lateral abscess /parietal abscess)
Depending on location :
- Gingival
- Periodontal (Acute / Chronic)
-Pericoronal
Etiology
[PERIODONTITIS RELATED]
- Extension of infection from PD pocket
- Lateral extension of inflammation
- Pocket with a tortuous course
- Incomplete removal of calculus
[NON PERIODONTITIS RELATED]
- Impaction of foreign bodies
- Endodontic perforation
- Lateral cyst infection
Signs and symptoms
Acute abscess
- Mild to severe discomfort
- Localized red, ovoid
- Mobility
- Tooth elevation in the socket
- Tenderness to percussion or biting
- Suppuration
- Localized inflammatory lesion
- Usually without systemic
- involvement
- Elevated temperature
Chronic abscess
- No pain or dull pain
- Periodontal pocket
- Slight tooth elevation
- Fistulous tract often associated with deep pocket
- Intermittent exudation
- Regional Lymphadnopathy
Enlargement in pregnancy
1. Marginal and generalised enlargement2. Single or multiple tumor like masses
Hormonal changes
Progesterone and estrogen
Vascular permeability — edema , t inflammatory response
Marginal enlargement
Generalised , more prominent interdentally
Bright red or magenta colour
Friable , smooth & shiny surface
Bleeding spontaneously on slight provocation
"Pregnancy rhinitis"
Tumor like gingival enlargement "Pregnancy tumor"
Discrete mushroom like ,flattened
spherical mass
Dusky red or magenta , smooth
glistening surface
Does not invade underlying bone
Semifirm — soft ., friable
sessile or pedunculated
Painless unless its size and shape foster
accumulation of debris
Treatment
• Removal of plaque and calculus
• Tumor like gingival enlargement is excised surgically.
Enlargement in puberty
• Male and female adoloscents
• Areas of plaque accumulation
• Facial surface
• Marginal and interdental
Enlargement in vitamin C deficiency
• Classic description of scurvy
• Acute deficiency — hemorrhage , collagen degeneration , edema
• modify response to plaque
Clinical features
- Bluish red , soft in texture, smooth & shiny surface
Haemorrhage — spontaneous / slight provocation
Surface necrosis with pseudomembrane formation
Plasma cell gingivitis
• Atypical gingivitis / plasma cell gingivostomatitis
• plasmacyte granuloma localised form • Allergic in origin
Clinical features
• Discrete tumor mass ,spherical in shape having pedunculated smooth surface
• it is either bright red or purple, can be friable or firm
• Painless
• Hemorrhage is characterisation.
Systemic Disease That Cause Gingival Enlargement
1. Leukemia
-malignant neoplasia of WBC precursors
- diffuse replacement of bone marrow proliferating leukemic cell
-abnormal number and forms of immature WBCs widespread infiltrates.
Clinical features
Diffuse / marginal
Localised / generalised
Overextension of gingiva
Discrete tumor like interproximal mass
Bluish red , shiny surface Firm
Hemorrhage
Leukemic infiltration
Leukemic cell infiltration of gingiva
Gingival thickness
Gingival pockets
Plaque accumulation
Secondary inflammatory lesion
2. Granulomatous disease a.Wegeners granulomatosis
- Acute granulomatous necrotising lesions of respiratory tract , nasal and oral defects
- Acute necrotising vasculitis
Clinical features
-oral mucosal ulcerations
-delayed healing
-Papillary enlargement reddish purple
-bleeds easily
-Strawberry gingiva
Etiology
- Unknown
- Immunologically mediated tissue injuries
NEOPLASTIC ENLARGEMENT
1. Benign tumors of gingiva Epulis
a. Fibroma
i) Giant cell fibroma
ii) Peripheral ossifying fibroma
b. Papilloma
- Proliferations of surface epithelium associated with HPV - HPV & 11
c. Peripheral Giant Cell Granuloma
d.Central Giant Cell Granuloma
- Arise within the jaw central cavitation
e. Leukoplakia
• WHO: White patch or plaque that does
not abrade & can't be diagnosed as the other disease
• Associated use of tobacco
Other probable factors: Candida, HPV-16, HPV-18 Trauma
2. Malignant tumors of gingiva
Squamous cell carcinoma:
• 90% of all Oral cancer
• 6th —most common cancer in males
• 12th - females
• Most common malignant tumor of gingiva
Malignant melanoma:
• Rare tumor hard palate, maxillary gingiva -older persons
• Darkly pigmented, rapid growth, early metastasis
Drugs associated with gingival overgrowth
Anticonvulsants
Phenytoin
Sodium valproate
Phenobarbitone
Vigabatrin
Immunosuppressants
Cyclosporin
Calcium channel blocker
Nifedipine
Felopdipine
Amlodipine
Phenylalkylamine
Benzothiazepine
Diltiazem
Idiopathic gingival enlargement
•Gingivomatosis , Elephantiasis, Idiopathic fibromatosis
• rare oral disease
• autosomal dominant
• hypertrichosis, mental retardation and epilepsy
• Nodular form
• Symmetric form- most common type
• During eruption of permanent teeth
• most common effects • diastemas,
• Malpositioning of teeth
• the alveolar bone is not affected (Bittencourt et al. 2000).
False enlargement
Commonly seen in 4 Paget's disease, Fibrous dysplasia, Cherubism, Central giant cell granuloma, Ameloblastoma, Osteoma and Osteosarcoma.
Dental hygiene is very important as it is the way that gives entry to microbials into our body. For systemic causes that leads to dental problems, cause must be treated first by medications. Medicines can be bought online from online pharmacy store in India.
Conclusion
Gingival enlargement are multifactorial and sophisticated in nature , which can be in respone to varied interaction between host and environment. GO considerably reduce the standard of life and should end in serios emotional and social problems thanks to esthetics and functionality hence the prevention and treatment supported the understanding the cause and underlying pathologic changes ,
Comments
Post a Comment